PhD position/Promotionsstelle in Biochemistry/Cell Biology

Mitochondria fulfil various essential functions for the survival of eukaryotic cell. Their biogenesis and functions depend on the import of more than 1000 different proteins that are produced as precursors on cytosolic ribosomes. The translocase of the outer mitochondrial membrane (TOM complex) forms the entry gate for preproteins, which are subsequently sorted via dedicated protein machineries into the different mitochondrial subcompartments. Mitochondrial protein import is an essential process for cell survival and defects cause severe neurodegenerative diseases and cardiomyopathy.

Activities and responsibilities

We investigate the mechanisms and quality control of protein biogenesis at the mitochondrial outer membrane. Particularly, dynamic protein-protein interactions in protein networks and their role in mitochondrial protein homeostastasis will be analysed.

Qualification profile

We are looking for highly motivated candidates with a Master degree in Biochemistry, Biology, Molecular Medicine, Chemistry or related fields. The University of Freiburg seeks to increase the number of female employees and therefore encourages qualified woman to apply for the position. Applicants with disabilities (Schwerbehinderte) will be given preferential consideration in case of equal qualification.

Benefits

PhD position (E13 E65%)

We use highly innovative techniques in biochemistry, molecular and cell biology such as protein-protein-interaction studies, activity assays, blue native electrophoresis and protein import and assembly assays. We offer a well-equipped workspace in active international team. The University of Freiburg provides an excellent scientific research environment.

Send application to

Applications including a CV, motivation letter, records of your Bachelor and Master studies and contact data for 2-3 references should be send via E-Mail to PD Dr. Thomas Becker: thomas.becker@biochemie.uni-freiburg.de.

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Institut für Biochemie und Molekularbiologie

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